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This secondary binding site may underlie some of the peptide's reported cytoprotective and anti-inflammatory properties observed in preclinical models, though the clinical significance remains under investigation
T.KizhakkedathuJ
doi:10.1038/s44259-024-00046-3 37
For instance, convincing in vitro work indicates that homocysteine may cause a specific impairment of the binding capacity of annexin II for t-PA that may alter the endothelial-dependent fibrinolytic potential (Hajjar et al, 1998)
It is often paired with regenerative therapies, such as stem cell treatments, to enhance recovery and outcomes
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