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The age-dependent decline of GHK-Cu levels provides a compelling rationale for supplementation as a strategy to restore regenerative functions diminished during aging

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Mitochondrial-mediated ROS cause oxidative damage to cellular structures, particularly mitochondrial DNA, somatic mutations, genome instability, inactivation of tumor suppressor genes, alterations in metabolic and signaling pathways with simultaneous activation of compensatory antioxidant mechanisms, all of which contribute to cell transformation and thus promote various types of cancer [42]

This blend is produced by our China-based factory to support laboratories that require consistent, high-purity material for tendon, ligament and muscle-related models