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However, when it was reported that GLP-1 was a substrate for DPP-4 in pharmacological in vitro kinetic studies (18), this was quickly followed by the demonstration that the metabolite generated by DPP-4 cleavage was the major circulating component of GLP-1-like immunoreactivity in healthy individuals (19) and that the same metabolite formed rapidly following exogenous administration of GLP-1 in both healthy subjects and those with T2DM (20)

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Unlike growth hormone secretagogues that rely on stimulating the pituitary first, IGF-1 LR3 works downstream at the tissue level

Inflammatory cytokine (interleukins 1, 6 and 8 and tumor necrosis factor-alpha) release from cultured human fetal membranes in response to endotoxic lipopolysaccharide mirrors amniotic fluid concentrations

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