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This hypothesis has a theoretical appeal, as the brain is considered particularly vulnerable to oxidative damage for several reasons
GSH is a key factor in redox signaling through its participation in trans hydrogenation reactions that remove hydrogen peroxide, free radicals, and other peroxides
When oxidative damage is being addressed not masked, not slowed marginally, but directly addressed at the cellular level function returns
Islet -cell failure in type 2 diabetesWithin the network of toxic lipids
As a classical inducer of ferroptosis, erastin can inhibit the transcriptional activity of AR and its splice variants in vitro and in vivo
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