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(2011) 17 independently demonstrated that N-acetylcysteine amide (NACA) remarkably protected retinal pigmented epithelium (RPE) and photoreceptors against oxidative cell damage and death, in vitro (ARPE-19 cell culture treated with tBHP) and in vivo (light damage mouse model)
It was designed to be a therapeutic agent for neurodegenerative diseases due to its potential to facilitate synaptogenesis, the formation of new synapses between neurons
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Improved physical performance: Animal studies show enhanced endurance and exercise capacity with MOTS-c treatment
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