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What they did: Multiple preclinical models (mice, rats) evaluated the combination AOD-9604 + GLP-1 agonist (Semaglutide or Liraglutide)

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Similar boosts in these same proteins were seen in the muscular tissue of murine models with critical limb ischemia once the research model was exposed to TB-500

BPC-157 and TB-500 operate through fundamentally different molecular mechanisms: BPC-157 modulates the nitric oxide system and upregulates growth factors including EGF, VEGF, and FGF-2 to promote cytoprotection and angiogenesis, while TB-500 sequesters G-actin monomers to regulate actin polymerization, cytoskeletal dynamics, and cell migration