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53 Therapeutic targeting of cardiac proteostasis The recognition that proteostasis failure contributes directly to cardiovascular disease has accelerated the development of therapeutic strategies aimed at restoring protein quality control rather than targeting isolated downstream manifestations
Adaptation to diverse nitrogen-limited environments by deletion or extrachromosomal element formation of the GAP1 locus
However, they work in different ways in the body
IV delivery achieves much higher blood levels and provides a more concentrated therapeutic effect
Cagrilintide has human weight-management research as a single compound
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