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USA 113 , E7490E7499 (2016)
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Importantly, these regions are also prone to the development of atherosclerosis [55,57-59]
Permanent inactivation of Huntingtons disease mutation by personalized allele-specific CRISPR/Cas9
Replacement effects are summarized in the first paragraph followed by our data interpretation in the second paragraph: Regression and pattern analyses revealed sequential kinetics for all mutants with an apparent common intersection point at the x axis (Supplementary Fig
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