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& Moyle, P
Individuals with ARTHS present with developmental delay, cognitive impairment (100% penetrance), delay or absence of speech (100%), facial dysmorphism (85%), feeding difficulties (78%), and neonatal hypotonia (76%) in a study of 76 individuals with KAT6A mutations (Kennedy et al, 2019)
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While only minimal amount of apoptosis upon treatment with Ru(II)-OH or NP2 was observed, the treatment with NP4 trigger high amounts of early and late apoptotic processes (flow cytometry plots: Fig
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