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The glucagon component increases energy expenditure and fat oxidation, potentially addressing the metabolic adaptation problem from yet another angle
Poe, F.L
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Front Public Health 13:1585711
Since Uch-L1 and UBB+1 exert opposite effects on A processing, metabolism and polyubiquitin chain formation, pharmacological modulation of UBB+1 or Uch-L1 levels in the brain may be a potential therapeutic strategy for AD
Biomarker assessment of serum and intracellular glutathione can reveal whether this depletion contributes to an individual's inflammatory burden
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