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We used thematic content analysis to extract information, and presented a narrative review account of the findings

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In summary, our study uncovered a novel role of adipocyte-expressed SIRT3 in mitigating infiltration and pro-inflammatory polarization of macrophages, thereby attenuating AT inflammation and protecting against obesity-related insulin resistance

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Taking a different approach, we then engineered the fusion molecule to create steric hindrance around the peptide and block its degradation by proteases either by creating an N-linked glycosylation site at position 25 of the GLP-1 analogue peptide (Ab#2_GLP1 W25N/V27S) or by introducing a disulphide bridge between the exendin-4 peptide and the linker (Ab#2_DSB#1)