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83 Gene therapies mediated by adeno-associated virus (AAV) targeting the key negative regulator Sav1 in the HippoYAP pathway can promote the nuclear translocation of YAP, thereby activating a series of genes related to cell proliferation and repair and enhancing the regenerative capacity of cardiomyocytes post-MI
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[13] [14] [15] Safety assessments also indicate a low risk of hypoglycemia and no significant elevation in cardiovascular or hepatic adverse events in non-diabetic populations across published studies
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