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doi: 10.1007/s10517-007-0159-y

Extensive persistent inflammation even when SARS-CoV-2-infected cells are only sporadically present at late stages of COVID-19 (Schurink et al., 2020) could also justify low GSH levels in association with over-abundance of cellular ROS, cellular oxidative damage, increased inflammation and cell death pathways activation seen in inflammaging (Zuo et al., 2019

PLoS ONE 4:e7316

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they may act as starting events in PD pathogenesis [22]