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They are heavily glycosylated 15,16 to resist microbial protease
Foxp1/4 control epithelial cell fate during lung development and regeneration through regulation of anterior gradient 2
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Furthermore, bile acid dysregulation, characterized by low conjugated primary bile acids and high hydrophobic secondary bile acids, stimulates FXR and TGR5 signaling pathways, exacerbating intestinal inflammation and modulating lipid metabolism [49]
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