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Its structure was derived from growth hormone releasing peptide-1 (GHRP-1) by removing the central Ala-Trp dipeptide and introducing aminoisobutyric acid (Aib) at the N-terminus for metabolic stability
Recently, attention has turned to using GLP-1 analogs, or GLP-1/glucagon co-agonism to treat these disorders [for review see (222228)]
Data from observational studies, particularly population-based cohorts, are most demonstrative of risk in a real-world scenario
In some cases, slowing the titration schedule under medical supervision can meaningfully reduce side-effect burden without compromising therapeutic benefit
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