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Treatment significantly reduced levels of pro-inflammatory cytokines including interleukin-1 (IL-1) and tumor necrosis factor- (TNF-) while increasing anti-inflammatory interleukin-10 (IL-10) expression
The peptide acts directly on fibroblasts at the repair site, and patients with tendinopathy generally report noticeable reduction in localized pain and improved range of motion within that window
(2022) Human umbilical cord mesenchymal stem cell-derived mitochondria (PN-101) attenuate LPS-induced inflammatory responses by inhibiting NFB signaling pathway
J Affect Disord (2014) 159:16
Animal studies, including rabbit models of segmental bone loss, have demonstrated improved regenerative outcomes following peptide administration
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