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Mechanistically, GHK-Cu preserved mitochondrial function by increasing mitochondrial membrane potential, alleviating age-related mitochondrial network fragmentation, shifting mitochondrial dynamics toward fusion via regulating drp-1 and fzo-1 expression, and promoting ATP biosynthesis

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In many clinical trials, no serious adverse reactions have been observed

Vitamin B12 also increases metabolic function and works synergistically with the MIC

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