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These two mechanisms are in line with the iron accumulation observed in various tissue samples of Fe-S cluster deficiency patients

It engages the same amylin/calcitonin receptors in the same hindbrain area postrema as native amylin and pramlintide, 5 11 and it drives the same vagally mediated slowing of the stomach that Samsom and colleagues quantified for pramlintide

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However, there are different ways to target the GLP-1 pathway, where two alternatives are stimulation of the receptor or the enhancing endogenous GLP-1 levels

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