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Dimethyl fumarate protects against hepatic ischemia-reperfusion injury by alleviating ferroptosis via the NRF2/SLC7A11/HO-1 axis
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[7] In fibroblast and endothelial-cell contexts, FAK-paxillin signaling provides a mechanistic framework for interpreting reported changes in adhesion turnover, cytoskeletal organization, and directional movement
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