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The anti-atherosclerotic effect of active forms was cancelled by exendin(939) or Pro3(GIP)specific antagonists for GLP-1 and GIP receptors
This mutation causes the PCD (Seth et al., 1999)
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At the end of the 20th century, endocrinologist Daniel Drucker was looking for a hormone similar enough to the human gut's GLP-1, which would have similar appetite-suppressing and blood sugar-regulating qualities, without being broken down by the human body so quickly
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